The polypeptide IGF-1 DES 1,3 is a peptide chain that consists of 67 different amino acids. It is considered to be highly anabolic in nature and it contains a molecular weight of 7371.4. It occasionally goes under the following alternate names:
Insulin-like growth factor 1
IGF-1 DES 1,3 and Specified Cellular Proliferation
It has been shown via scientific study based on animal test subjects that IGF-1 DES 1,3’s main means of functional mechanics can be inherently linked to a highly specified form of cellular proliferation known as hyperplasia, although it can sometimes be known as hypergenesis. In essence, this condition is a process that describes the regulation of the increase or proliferation of cells.
There are several triggers that can lead to hyperplasia to be created. The first trigger involves an increased demand for cells in a given area in order to promote stabilization in a region where cellular proliferation is needed, such as when a base layer of epidermis is needed in order to compensate for a natural loss of skin. Another trigger for such proliferation may be to aid in the holding off of one of several forms of ailments that could impede the proper performance of an animal test subject’s endocrine system. Or the peptide could induce the onset of hyperplasia as a chronic inflammatory response in order to fight off the process of inflammation in a given segment of the body. Hyperplasia may also be initiated via the peptide to protect against other forms of cellular loss that was brought about by damage or disease throughout the body.
Scientific study that has been built on animal test subjects has zeroed in on IGF-1 DES 1,3’s capacity to create hyperplasia to determine that the peptide ultimately has the ability to control the proliferation of cells and the development of tissue.
IGF-1 DES 1,3 and Neurological Growth
Additional studies have also included research on its possible ability to effect neurological growth, as they notate that it has demonstrated an ability to influence neuronal structure and functionality through the entire lifespan of an animal test subject. Furthermore, these studies have shown that it has a capacity to promote a state of neuroregenesis; a process in which nerve tissue growth can be promoted. Ultimately, this connection has led scientific study based on animal test subjects to determine that IGF-1 DES 1,3 could function as a possible aid in the treatment of a host of injuries that could negatively impact the nervous system. These injuries would include instances that relate to neuropraxia, axonotmesis, and neurotmesis.
Positive Results of IGF-1 DES 1,3’s Hyperplasia Influence
Scientific study that has been based on animal test subjects has shown that IGF-1 DES 1,3’s ability to create hyperplasia can be linked to several hypothesized benefits.
The first of these benefits has been determined to be linked to a possible acceleration of muscle repair. Because of the mechanics of IGF-1 DES 1,3 and its faculty to promote hyperplasia, scientific study that has been based on animal test subjects has determined that it can boost the type of cellular proliferation that is required in order to repair muscle tissue. This process can eventually lead to a boost in muscle growth. This derived benefit can also be somewhat tied to the peptide’s highly anabolic qualities.
Another benefit that has been linked to IGF-1 DES 1,3 is in relation to slowing down the overall aging process. Specifically, scientific study on laboratory rats has been able to determine that IGF-1 DES 1,3’s promotion of cell proliferation slowed down the type of naturally occurring degradation of muscle fibers that control the elastic and flexible properties of the skin and muscles. This promotion allowed middle-aged and older rats to maintain a consistent level of power and speed that was similar in nature to younger rats.
A third benefit that has been derived via scientific study on animal test subjects is tied to IGF-1 DES 1,3’s ability to promote an increased rate of injury recovery. These studies have indicated hat IGF-1 DES 1,3’s capacity to boost cellular proliferation can cause a boosted amount of cells to be produced in times when an animal test subject suffers an injury. By that rationale, it has been determined that this proliferation can cause a much faster rate of healing to ensue.